Journal of Pain, Vertigo and Headache
JPHV - Journal of Pain, Headache and Vertigo is a peer-reviewed and open access journal that focuses on promoting pain, headache and vertigo. This journal publishes original articles, reviews, and also interesting case reports. JPHV - Journal of Pain, Headache and Vertigo is an international scientific journal, published twice a year by PERDOSSI (Perhimpunan Dokter Spesialis Saraf Indonesia Cabang Malang) - Indonesian Neurological Association Branch of Malang cooperated with Neurology Residency Program, Faculty of Medicine Brawijaya University, Malang, Indonesia.
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<![CDATA[CLUSTER HEADACHE ]]>
<![CDATA[Michelle Anisa]]>;
<![CDATA[Shahdevi Nandar Kurniawan]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
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DOI: 10.21776/ub.jphv.2022.003.02.1
<![CDATA[Cluster headache (CH) is a trigeminal autonomic cephalgia characterized by attacks of severe unilateral headache accompanied by ipsilateral autonomic symptoms. The prevalence of cluster headache in the overall population is 1 in every 1000 people. The exact etiology of cluster headache remains unclear. However, it is thought that there is a connection between the trigeminovascular system, parasympathetic nerve fibers involved in trigeminal autonomic reflexes, and the hypothalamus. Treatment of CH has three stages, namely: abortive, transitional, and preventive. Cluster headaches tend to subside with age with less frequent attacks and longer periods of remission between attacks.]]>
<![CDATA[CLASSICAL MIGRAINE ]]>
<![CDATA[Shahdevi Nandar Kurniawan]]>;
<![CDATA[Dyah Kusuma Wardhani]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
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DOI: 10.21776/ub.jphv.2022.003.02.2
<![CDATA[A classic migraine is a recurrent attack of visual, sensory, or other central nervous system symptoms that are unilateral and last several minutes, followed or not followed by a migraine attack. Migraine commonly occurs in 19% of women and 11% of men worldwide, with 20% of sufferers experiencing classic migraine. The etiopathophysiology of classical migraine is not known with certainty, but vascular, neurological, and genetic dysfunction are suspected to be the cause. Classical migraine pathophysiology is associated with the theory of cortical spreading depression, which can explain the process of aura. There are four phases in classical migraine, namely prodromal, aura, headache, and prodromal phases, each of which has its own symptoms. This is the basis for the diagnosis of migraine, which is established based on the history and physical examination. Migraine therapy includes preventive therapy (lifestyle changes and prophylactic administration) as well as abortive therapy (administration of specific and non-specific drugs).]]>
<![CDATA[TENSION TYPE HEADACHE (TTH) ]]>
<![CDATA[Auliya Nur Muthmainnina]]>;
<![CDATA[Shahdevi Nandar Kurniawan]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
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DOI: 10.21776/ub.jphv.2022.003.02.3
<![CDATA[Tension Type Headache (TTH) is the most common type of headache in all age groups worldwide. Because of its high prevalence and possible association with medical and psychiatric comorbidities, TTH has a large socioeconomic impact. TTH is the type of headache that most patients suffer from, ranging from mild to severe pain that reduces their ability to carry out daily activities. TTH can be classified into an episodic tension-type headache (ETTH) and chronic tension-type headache (CTTH). The lifetime prevalence of TTH is high (78%). Approximately 24% to 37% experience TTH several times a month, 10% experience weekly and 2% to 3% of the population have chronic TTH disease. TTH treatment is carried out with pharmacological and non-pharmacological approaches.]]>
<![CDATA[PERSISTENT HEADACHE AFTER CEREBELLUM HEMORRHAGE STROKE ]]>
<![CDATA[Nata Sanjaya]]>;
<![CDATA[Shahdevi Nandar Kurniawan]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
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DOI: 10.21776/ub.jphv.2022.003.02.4
<![CDATA[Cerebrovascular disease is the number one cause of epilepsy in the elderly population. Headaches are relatively common in patients with cerebrovascular disorders. The frequency of stroke-related headaches ranges from 7% to 65% with different types of headaches. The prevalence of persistent post-stroke headaches from 7-23%, with follow-up times ranging from 3 months to 3 years after stroke. Persistent headache in the population was associated with high depression and fatigue scores and significantly impacted returning to work. Most headaches at stroke onset will resolve, persistent headaches are a real entity even years after the stroke. The mechanism that might explain the relationship between headache and hemorrhagic stroke is still unclear, including changes in blood vessel walls supported by endothelial dysfunction in migraine sufferers as well as comorbid vascular risk factors such as arterial hypertension or platelet dysfunction. Headache after stroke intracerebral hemorrhage is believed to be the result of vasoconstriction that causes ischemia of the vessel wall.]]>
<![CDATA[COMPARISON OF HYDRODISSECTION INJECTION THERAPY USING ULTRASONOGRAPHIC AS GUIDES BETWEEN TRIAMCINOLONE ACETONIDE AND 5% DEXTROSE IN CARPAL TUNNEL SYNDROME ]]>
<![CDATA[Widodo Mardi Santoso]]>;
<![CDATA[Ika Sedar Wasis Sasono]]>;
<![CDATA[Catur Ari Setianto]]>;
<![CDATA[Nuretha Hevy]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
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DOI: 10.21776/ub.jphv.2022.003.02.5
<![CDATA[Background: Carpal Tunnel Syndrome (CTS) is a symptomatic compression neuropathy of the median nerve characterized by increased pressure in the carpal tunnel and decreased nerve function due to compression of the median nerve in the carpal tunnel. The purpose of the hydrodissection injection method in CTS is to separate the soft tissue adhesions that cause nerve compression and this method are known for being minimally invasive, fast healing, and easy to apply. Local injection of triamcinolone acetonide (TCA) is often used as therapy for CTS because it stabilizes the sodium channels and reduces abnormal stimulatio, thus it relieved the pain. 5% dextrose injection (D5W) is also widely used as therapy of CTS because it is harmless to nerves and may reduce neurogenic inflammation through inhibition of capsaicin-sensitive receptors. Aim: To compare the effectivity of hydrodissection injection therapy using ultrasound guidance with triamcinolone acetonide and 5% dextrose in CTS. Methods: This study recruited 30 participants who diagnosed with CTS and met the inclusion criteria. Participants were divided into two treatment groups, the first group (n=15) was given 1ml TCA injection and 1 ml lidocaine 2%, while the second group (n=15) was given 5% 5 ml Dextrose injection. The parameters measured in this study were NRS, FSS, and SSS value before injection and 4 weeks after injection of the agent. We compared these parameters at week four after injection between the TCA group and the D5W group. Results: NRS values before and 4 weeks after TCA injection (sig 0.001; p <0.05), FSS values (sig 0.020; p <0.05), and SSS values (sig 0.001; p <0.05). NRS before and 4 weeks after injection of D5W (sig 0.002; p <0.05), FSS (sig 0.001; p <0.05), and SSS (sig 0.000; p <0.05). Comparison between TCA injection and D5W injection at 4 weeks after the injection showed that the results was significantly different on NRS (sig 0.806; p> 0.05) for FSS (sig 0.512; p> 0.05) and SSS (sig 0.293; p> 0.05). Conclusion: There is a significant difference in NRS, FSS and SSS values at 4 weeks after hydrodissection injection, using either TCA or D5W. TCA hydrodissection injection compared to D5W hydrodissection injection was equally effective in improving NRS, FSS and SSS after 4 weeks of injection.]]>
<![CDATA[CLUSTER HEADACHE ]]>
<![CDATA[Michelle Anisa]]>;
<![CDATA[Shahdevi Nandar Kurniawan]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
Show Abstract
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DOI: 10.21776/ub.jphv.2022.003.02.1
<![CDATA[Cluster headache (CH) is a trigeminal autonomic cephalgia characterized by attacks of severe unilateral headache accompanied by ipsilateral autonomic symptoms. The prevalence of cluster headache in the overall population is 1 in every 1000 people. The exact etiology of cluster headache remains unclear. However, it is thought that there is a connection between the trigeminovascular system, parasympathetic nerve fibers involved in trigeminal autonomic reflexes, and the hypothalamus. Treatment of CH has three stages, namely: abortive, transitional, and preventive. Cluster headaches tend to subside with age with less frequent attacks and longer periods of remission between attacks.]]>
<![CDATA[CLASSICAL MIGRAINE ]]>
<![CDATA[Shahdevi Nandar Kurniawan]]>;
<![CDATA[Dyah Kusuma Wardhani]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
Show Abstract
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DOI: 10.21776/ub.jphv.2022.003.02.2
<![CDATA[A classic migraine is a recurrent attack of visual, sensory, or other central nervous system symptoms that are unilateral and last several minutes, followed or not followed by a migraine attack. Migraine commonly occurs in 19% of women and 11% of men worldwide, with 20% of sufferers experiencing classic migraine. The etiopathophysiology of classical migraine is not known with certainty, but vascular, neurological, and genetic dysfunction are suspected to be the cause. Classical migraine pathophysiology is associated with the theory of cortical spreading depression, which can explain the process of aura. There are four phases in classical migraine, namely prodromal, aura, headache, and prodromal phases, each of which has its own symptoms. This is the basis for the diagnosis of migraine, which is established based on the history and physical examination. Migraine therapy includes preventive therapy (lifestyle changes and prophylactic administration) as well as abortive therapy (administration of specific and non-specific drugs).]]>
<![CDATA[TENSION TYPE HEADACHE (TTH) ]]>
<![CDATA[Auliya Nur Muthmainnina]]>;
<![CDATA[Shahdevi Nandar Kurniawan]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
Show Abstract
|
Download Original
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Original Source
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Check in Google Scholar
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DOI: 10.21776/ub.jphv.2022.003.02.3
<![CDATA[Tension Type Headache (TTH) is the most common type of headache in all age groups worldwide. Because of its high prevalence and possible association with medical and psychiatric comorbidities, TTH has a large socioeconomic impact. TTH is the type of headache that most patients suffer from, ranging from mild to severe pain that reduces their ability to carry out daily activities. TTH can be classified into an episodic tension-type headache (ETTH) and chronic tension-type headache (CTTH). The lifetime prevalence of TTH is high (78%). Approximately 24% to 37% experience TTH several times a month, 10% experience weekly and 2% to 3% of the population have chronic TTH disease. TTH treatment is carried out with pharmacological and non-pharmacological approaches.]]>
<![CDATA[PERSISTENT HEADACHE AFTER CEREBELLUM HEMORRHAGE STROKE ]]>
<![CDATA[Nata Sanjaya]]>;
<![CDATA[Shahdevi Nandar Kurniawan]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
Show Abstract
|
Download Original
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Original Source
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Check in Google Scholar
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DOI: 10.21776/ub.jphv.2022.003.02.4
<![CDATA[Cerebrovascular disease is the number one cause of epilepsy in the elderly population. Headaches are relatively common in patients with cerebrovascular disorders. The frequency of stroke-related headaches ranges from 7% to 65% with different types of headaches. The prevalence of persistent post-stroke headaches from 7-23%, with follow-up times ranging from 3 months to 3 years after stroke. Persistent headache in the population was associated with high depression and fatigue scores and significantly impacted returning to work. Most headaches at stroke onset will resolve, persistent headaches are a real entity even years after the stroke. The mechanism that might explain the relationship between headache and hemorrhagic stroke is still unclear, including changes in blood vessel walls supported by endothelial dysfunction in migraine sufferers as well as comorbid vascular risk factors such as arterial hypertension or platelet dysfunction. Headache after stroke intracerebral hemorrhage is believed to be the result of vasoconstriction that causes ischemia of the vessel wall.]]>
<![CDATA[COMPARISON OF HYDRODISSECTION INJECTION THERAPY USING ULTRASONOGRAPHIC AS GUIDES BETWEEN TRIAMCINOLONE ACETONIDE AND 5% DEXTROSE IN CARPAL TUNNEL SYNDROME ]]>
<![CDATA[Widodo Mardi Santoso]]>;
<![CDATA[Ika Sedar Wasis Sasono]]>;
<![CDATA[Catur Ari Setianto]]>;
<![CDATA[Nuretha Hevy]]>
<![CDATA[ Journal of Pain, Headache and Vertigo Vol. 3 No. 2 (2022): September ]]>
Publisher : <![CDATA[Journal of Pain, Headache and Vertigo ]]>
Show Abstract
|
Download Original
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Original Source
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Check in Google Scholar
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DOI: 10.21776/ub.jphv.2022.003.02.5
<![CDATA[Background: Carpal Tunnel Syndrome (CTS) is a symptomatic compression neuropathy of the median nerve characterized by increased pressure in the carpal tunnel and decreased nerve function due to compression of the median nerve in the carpal tunnel. The purpose of the hydrodissection injection method in CTS is to separate the soft tissue adhesions that cause nerve compression and this method are known for being minimally invasive, fast healing, and easy to apply. Local injection of triamcinolone acetonide (TCA) is often used as therapy for CTS because it stabilizes the sodium channels and reduces abnormal stimulatio, thus it relieved the pain. 5% dextrose injection (D5W) is also widely used as therapy of CTS because it is harmless to nerves and may reduce neurogenic inflammation through inhibition of capsaicin-sensitive receptors. Aim: To compare the effectivity of hydrodissection injection therapy using ultrasound guidance with triamcinolone acetonide and 5% dextrose in CTS. Methods: This study recruited 30 participants who diagnosed with CTS and met the inclusion criteria. Participants were divided into two treatment groups, the first group (n=15) was given 1ml TCA injection and 1 ml lidocaine 2%, while the second group (n=15) was given 5% 5 ml Dextrose injection. The parameters measured in this study were NRS, FSS, and SSS value before injection and 4 weeks after injection of the agent. We compared these parameters at week four after injection between the TCA group and the D5W group. Results: NRS values before and 4 weeks after TCA injection (sig 0.001; p <0.05), FSS values (sig 0.020; p <0.05), and SSS values (sig 0.001; p <0.05). NRS before and 4 weeks after injection of D5W (sig 0.002; p <0.05), FSS (sig 0.001; p <0.05), and SSS (sig 0.000; p <0.05). Comparison between TCA injection and D5W injection at 4 weeks after the injection showed that the results was significantly different on NRS (sig 0.806; p> 0.05) for FSS (sig 0.512; p> 0.05) and SSS (sig 0.293; p> 0.05). Conclusion: There is a significant difference in NRS, FSS and SSS values at 4 weeks after hydrodissection injection, using either TCA or D5W. TCA hydrodissection injection compared to D5W hydrodissection injection was equally effective in improving NRS, FSS and SSS after 4 weeks of injection.]]>
